Tiny sacs from embryonic stem cells kept older cells from aging in the lab, according to Cornell Chronicle.
Cornell researchers found that extracellular vesicles stopped cells from entering senescence—when cells stop dividing—by triggering a protein pathway that blocks oxidative stress. The key: a protein called fibronectin that kicks off anti-aging signals once the vesicles latch onto cells.
Next, the team plans to test the vesicles in mice and, eventually, explore their use in reprogrammed human cells to boost healthy aging.
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